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glutathione and breast cancer risk An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in
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glutathione and breast cancer risk An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in
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Frontiers Glutathione responsive and exhausting metal nanomedicines for robust synergistic cancer therapy What do cancer cells eat? New research reveals surprising fuel source International Journal of Oncology Association of glutathione S transferase T1, M1, and P1 polymorphisms TCRM Dove Medical Press Glutathione Dependent Pathways in Cancer Cells
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glutathione and breast cancer risk An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in
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